MiR-200c is a cMyc-activated miRNA that promotes nasopharyngeal carcinoma by downregulating PTEN

نویسندگان

  • Pan Chen
  • Xiaofang Guo
  • Liming Zhang
  • Wenling Zhang
  • Qingyu Zhou
  • Zhi Tian
  • Ying Zheng
  • Qianjin Liao
  • Heran Wang
  • Guiyuan Li
  • Jin Huang
  • Xiayu Li
چکیده

The c-Myc transcription factor regulates a complex transcriptional program that leads to cellular transformation by targeting a large number of protein-encoding genes and non-coding RNAs. In this study, we show that a microRNA, miR-200c, is a novel c-Myc target that promotes cellular transformation and metastasis in nasopharyngeal carcinoma. MiR-200c achieves this oncogenic effect, at least in part, by targeting and inhibiting the tumor suppressor gene PTEN (phosphatase and tensin homolog), which is a key inhibitor of the AKT kinase signaling that promotes tumorigenesis in nasopharyngeal carcinoma. Our study thus identifies cMyc-miR-200c-PTEN-AKT as a functional module that promotes cellular transformation in nasopharyngeal carcinoma.

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عنوان ژورنال:

دوره 8  شماره 

صفحات  -

تاریخ انتشار 2017